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Submitted: 29 Sep 2025
Revision: 01 Feb 2026
Accepted: 21 Feb 2026
ePublished: 31 Mar 2026
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Pharm Sci. Inpress.
doi: 10.34172/PS.026.43308
  Abstract View: 13

Original Article

Enhancing Radiosensitivity in Prostate Adenocarcinoma by 7-Geranyloxycoumarin: The Potential Roles of β-Catenin, c-MYC, and PSMD10

Nafiseh Bagheri ORCID logo, Yasaman Abolhassani ORCID logo, Fatemeh Behnam Rassouli ORCID logo, Khadijeh Jamialahmadi* ORCID logo
*Corresponding Author: Email: jamialahmadikh@mums.ac.ir

Abstract

Background: Prostate adenocarcinoma is commonly treated with radiation and chemotherapy, but resistance and toxicity limit their success, highlighting the need for novel radiosensitizers. We investigated the effects of 7-Geranyloxycoumarin, alone and in combination with radiation, on the expression of β-catenin (CTNNB1), c-MYC, and Gankyrin (PSMD10) in prostate adenocarcinoma cells. Methods: Interactome mapping and enrichment analysis were performed using STRING. To validate their expression in prostate adenocarcinoma samples, the GEPIA2 database was used. For in vitro analysis, human prostate carcinoma PC-3 cells were pretreated with 40 µM 7-Geranyloxycoumarin and exposed to 4 Gy radiation. After 72 h, real-time PCR was performed to assess gene expression. Results: In silico analysis confirmed the interaction between CTNNB1, MYC, and PSMD10, and revealed their overexpression in prostate adenocarcinoma samples. In PC-3 cells, 7-Geranyloxycoumarin treatment significantly decreased c-MYC expression (p < 0.0001). Radiation alone and DMSO-treated irradiated cells also showed a significant reduction in c-MYC expression (p < 0.001). Conversely, combined treatment significantly increased CTNNB1 expression (p < 0.01) and decreased PSMD10 expression. Conclusion: Our study demonstrated elevated expression of CTNNB1 and decrease of c-MYC, and PSMD10 in prostate adenocarcinoma, and highlighted that 7-Geranyloxycoumarin, alone or in combination with radiation, markedly modulates their expression in PC-3 cells.
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